Hannah Jones
University of Bristol
H-index: 22
Europe-United Kingdom
About Hannah Jones
Hannah Jones, With an exceptional h-index of 22 and a recent h-index of 21 (since 2020), a distinguished researcher at University of Bristol,
His recent articles reflect a diverse array of research interests and contributions to the field:
Precursors and correlates of transient and persistent longitudinal profiles of psychotic experiences from late childhood through early adulthood
Mendelian randomisation for psychiatry: how does it work, and what can it tell us?
Examining pathways between genetic liability for schizophrenia and patterns of tobacco and cannabis use in adolescence
Parental inflammatory bowel disease and autism in the offspring: Triangulating the evidence using four complementary study designs
Genetic liability to schizophrenia is associated with exposure to traumatic events in childhood
Causal associations between fatty acid measures and schizophrenia-a two-sample Mendelian randomization study
Childhood trauma as a mediator of the association between autistic traits and psychotic experiences: evidence from the ALSPAC birth cohort
Plasma polyunsaturated fatty acids and mental disorders in adolescence and early adulthood: cross-sectional and longitudinal associations in a general population cohort
Hannah Jones Information
University | University of Bristol |
---|---|
Position | ___ |
Citations(all) | 2619 |
Citations(since 2020) | 2289 |
Cited By | 1166 |
hIndex(all) | 22 |
hIndex(since 2020) | 21 |
i10Index(all) | 29 |
i10Index(since 2020) | 29 |
University Profile Page | University of Bristol |
Top articles of Hannah Jones
Precursors and correlates of transient and persistent longitudinal profiles of psychotic experiences from late childhood through early adulthood
Authors
Alexandros Rammos,Sarah A Sullivan,Daphne Kounali,Hannah J Jones,Gemma Hammerton,Lindsey A Hines,Glyn Lewis,Peter B Jones,Mary Cannon,Andrew Thompson,Dieter Wolke,Jon Heron,Stanley Zammit
Journal
The British Journal of Psychiatry
Published Date
2022/6
Background Psychotic experiences are reported by 5–10% of young people, although only a minority persist and develop into psychotic disorders. It is unclear what characteristics differentiate those with transient psychotic experiences from those with persistent psychotic experiences that are more likely to be of clinical relevance. Aims To investigate how longitudinal profiles of psychotic experiences, created from assessments at three different time points, are influenced by early life and co-occurring factors. Method Using data from 8045 individuals from a birth cohort study, longitudinal profiles of psychotic experiences based on semi-structured interviews conducted at 12, 18 and 24 years were defined. Environmental, cognitive, psychopathological and genetic determinants of these profiles were investigated, along with concurrent changes in psychopathology and cognition. Results Following multiple …
Mendelian randomisation for psychiatry: how does it work, and what can it tell us?
Authors
Robyn E Wootton,Hannah J Jones,Hannah M Sallis
Published Date
2022/1
The successful prevention of mental illness relies upon the identification of causal, modifiable risk factors. However, observational evidence exploring such risk factors often produces contradictory results and randomised control trials are often expensive, time-consuming or unethical to conduct. Mendelian randomisation (MR) is a complementary approach that uses naturally occurring genetic variation to identify possible causal effects between a risk factor and an outcome in a time-efficient and low-cost manner. MR utilises genetic variants as instrumental variables for the risk factor of interest. MR studies are becoming more frequent in the field of psychiatry, warranting a reflection upon both the possibilities and the pitfalls. In this Perspective, we consider several limitations of the MR method that are of particular relevance to psychiatry. We also present new MR methods that have exciting applications to questions of …
Examining pathways between genetic liability for schizophrenia and patterns of tobacco and cannabis use in adolescence
Authors
Hannah J Jones,Gemma Hammerton,Tayla McCloud,Lindsey A Hines,Caroline Wright,Suzanne H Gage,Peter Holmans,Peter B Jones,George Davey Smith,David EJ Linden,Michael C O'Donovan,Michael J Owen,James T Walters,Marcus R Munafò,Jon Heron,Stanley Zammit
Journal
Psychological medicine
Published Date
2022/1
BackgroundIt is not clear to what extent associations between schizophrenia, cannabis use and cigarette use are due to a shared genetic etiology. We, therefore, examined whether schizophrenia genetic risk associates with longitudinal patterns of cigarette and cannabis use in adolescence and mediating pathways for any association to inform potential reduction strategies.MethodsAssociations between schizophrenia polygenic scores and longitudinal latent classes of cigarette and cannabis use from ages 14 to 19 years were investigated in up to 3925 individuals in the Avon Longitudinal Study of Parents and Children. Mediation models were estimated to assess the potential mediating effects of a range of cognitive, emotional, and behavioral phenotypes.ResultsThe schizophrenia polygenic score, based on single nucleotide polymorphisms meeting a training-set p threshold of 0.05, was associated with late-onset …
Parental inflammatory bowel disease and autism in the offspring: Triangulating the evidence using four complementary study designs
Authors
Aws Sadik,Christina Dardani,Panagiota Pagoni,Alexandra Havdahl,Evie Stergiakouli,iPSYCH Autism Spectrum Disorder Working Group Grove Jakob 14 15 16 17,Golam M Khandaker,Sarah A Sullivan,Stan Zammit,Hannah J Jones,George Davey Smith,Christina Dalman,Håkan Karlsson,Renee M Gardner,Dheeraj Rai
Journal
Nature Medicine
Published Date
2022/7
Evidence linking parental inflammatory bowel disease (IBD) with autism in children is inconclusive. We conducted four complementary studies to investigate associations between parental IBD and autism in children, and elucidated their underlying etiology. Conducting a nationwide population-based cohort study using Swedish registers, we found evidence of associations between parental diagnoses of IBD and autism in children. Polygenic risk score analyses of the Avon Longitudinal Study of Parents and Children suggested associations between maternal genetic liability to IBD and autistic traits in children. Two-sample Mendelian randomization analyses provided evidence of a potential causal effect of genetic liability to IBD, especially ulcerative colitis, on autism. Linkage disequilibrium score regression did not indicate a genetic correlation between IBD and autism. Triangulating evidence from these four …
Genetic liability to schizophrenia is associated with exposure to traumatic events in childhood
Authors
Hannah M Sallis,Jazz Croft,Alexandra Havdahl,Hannah J Jones,Erin C Dunn,George Davey Smith,Stanley Zammit,Marcus R Munafò
Journal
Psychological medicine
Published Date
2021/8
BackgroundThere is a wealth of literature on the observed association between childhood trauma and psychotic illness. However, the relationship between childhood trauma and psychosis is complex and could be explained, in part, by gene–environment correlation.MethodsThe association between schizophrenia polygenic scores (PGS) and experiencing childhood trauma was investigated using data from the Avon Longitudinal Study of Parents and Children (ALSPAC) and the Norwegian Mother, Father and Child Cohort Study (MoBa). Schizophrenia PGS were derived in each cohort for children, mothers, and fathers where genetic data were available. Measures of trauma exposure were derived based on data collected throughout childhood and adolescence (0–17 years; ALSPAC) and at age 8 years (MoBa).ResultsWithin ALSPAC, we found a positive association between schizophrenia PGS and exposure to …
Causal associations between fatty acid measures and schizophrenia-a two-sample Mendelian randomization study
Authors
Hannah J Jones,Maria Carolina Borges,Rebecca Carnegie,David Mongan,Peter J Rogers,Sarah J Lewis,Andrew D Thompson,Stanley Zammit
Journal
medRxiv
Published Date
2021/3/17
ObjectiveAlthough studies suggest that erythrocyte concentrations of omega-3 and omega-6 fatty acids are lower in individuals with schizophrenia, evidence of beneficial effects of omega-3 fatty acid supplementation is limited. This study therefore aimed to determine whether omega-3 and omega-6 fatty acid levels are causally related to schizophrenia.MethodsCausality was evaluated using the inverse variance weighted (IVW) 2-sample Mendelian randomization (MR) method using fatty acid levels and schizophrenia genome-wide association study results. Weighted median, weighted mode, and MR Egger regression methods were used as sensitivity analyses. To address the mechanism, analyses were performed using instruments within the FADS and ELOVL2 genes. Multivariable MR (MVMR) was used to estimate direct effects of omega-3 fatty acids on schizophrenia, independent of omega-6 fatty acids, lipoproteins and triglycerides.ResultsMR analyses indicated that long-chain omega-3 and omega-6 fatty acid levels were associated with lower risk of schizophrenia (docosahexaenoic acid [DHA] ORIVW: 0.83, 95% CI: 0.75-0.92). In contrast, short-chain fatty acids were associated with an increased risk of schizophrenia (alpha-linolenic acid ORIVW: 1.07, 95% CI: 0.98-1.18). Causal effects were consistent across sensitivity and FADS single-SNP analyses. MVMR indicated that the protective effect of DHA on schizophrenia persisted after conditioning on other lipids (ORIVW: 0.84, 95% CI: 0.71-1.01).ConclusionsResults are consistent with protective effects of long-chain omega-3 and omega-6 fatty acids on schizophrenia suggesting that people …
Childhood trauma as a mediator of the association between autistic traits and psychotic experiences: evidence from the ALSPAC birth cohort
Authors
Christina Dardani,Rik Schalbroeck,Paul Madley-Dowd,Hannah J Jones,Daniela Strelchuk,Gemma Hammerton,Jazz Croft,Sarah A Sullivan,Stan Zammit,Jean-Paul Selten,Dheeraj Rai
Journal
Schizophrenia Bulletin
Published Date
2023/3/1
Background Little is known on whether associations between childhood autistic traits and psychotic experiences persist into adulthood and whether genetic confounding and childhood trauma influence them. Here we investigate the associations between childhood autistic traits and psychotic experiences until young adulthood and assess the influence of schizophrenia polygenic risk and childhood traumatic experiences, using the Avon Longitudinal Study of Parents and Children (ALSPAC) population-based birth cohort. Study design We used a measure of broad autistic traits (autism factor mean score), and four dichotomised measures of autistic traits capturing social communication difficulties (age 7), repetitive behaviours (age 5), sociability (age 3), and pragmatic language (age 9). Psychotic experiences were assessed at ages 18 and 24 using the semi-structured Psychosis …
Plasma polyunsaturated fatty acids and mental disorders in adolescence and early adulthood: cross-sectional and longitudinal associations in a general population cohort
Authors
David Mongan,Colm Healy,Hannah J Jones,Stan Zammit,Mary Cannon,David R Cotter
Journal
Translational Psychiatry
Published Date
2021/5/31
Polyunsaturated fatty acids (PUFAs) may be pertinent to the development of mental disorders, for example via modulation of inflammation and synaptogenesis. We wished to examine cross-sectional and longitudinal associations between PUFAs and mental disorders in a large cohort of young people. Participants in the Avon Longitudinal Study of Parents and Children were interviewed and provided blood samples at two sampling periods when approximately 17 and 24 years old. Plasma PUFA measures (total omega-6 [n-6], total omega-3 [n-3], n-6:n-3 ratio and docosahexaenoic acid [DHA] percentage of total fatty acids) were assessed using nuclear magnetic resonance spectroscopy. Cross-sectional and longitudinal associations between standardised PUFA measures and three mental disorders (psychotic disorder, moderate/severe depressive disorder and generalised anxiety disorder [GAD]) were measured …
The potential shared role of inflammation in insulin resistance and schizophrenia: A bidirectional two-sample mendelian randomization study
Authors
Benjamin I Perry,Stephen Burgess,Hannah J Jones,Stan Zammit,Rachel Upthegrove,Amy M Mason,Felix R Day,Claudia Langenberg,Nicholas J Wareham,Peter B Jones,Golam M Khandaker
Journal
PLoS medicine
Published Date
2021/3/12
Background Insulin resistance predisposes to cardiometabolic disorders, which are commonly comorbid with schizophrenia and are key contributors to the significant excess mortality in schizophrenia. Mechanisms for the comorbidity remain unclear, but observational studies have implicated inflammation in both schizophrenia and cardiometabolic disorders separately. We aimed to examine whether there is genetic evidence that insulin resistance and 7 related cardiometabolic traits may be causally associated with schizophrenia, and whether evidence supports inflammation as a common mechanism for cardiometabolic disorders and schizophrenia. Methods and findings We used summary data from genome-wide association studies of mostly European adults from large consortia (Meta-Analyses of Glucose and Insulin-related traits Consortium (MAGIC) featuring up to 108,557 participants; Diabetes Genetics Replication And Meta-analysis (DIAGRAM) featuring up to 435,387 participants; Global Lipids Genetics Consortium (GLGC) featuring up to 173,082 participants; Genetic Investigation of Anthropometric Traits (GIANT) featuring up to 339,224 participants; Psychiatric Genomics Consortium (PGC) featuring up to 105,318 participants; and Cohorts for Heart and Aging Research in Genomic Epidemiology (CHARGE) consortium featuring up to 204,402 participants). We conducted two-sample uni- and multivariable mendelian randomization (MR) analysis to test whether (i) 10 cardiometabolic traits (fasting insulin, high-density lipoprotein and triglycerides representing an insulin resistance phenotype, and 7 related cardiometabolic traits: low …
An exploration of the genetic epidemiology of non-suicidal self-harm and suicide attempt
Authors
Abigail Emma Russell,Gibran Hemani,Hannah J Jones,Tamsin Ford,David Gunnell,Jon Heron,Carol Joinson,Paul Moran,Caroline Relton,Matthew Suderman,Sarah Watkins,Becky Mars
Journal
BMC psychiatry
Published Date
2021/4/23
BackgroundEmpirical evidence supporting the distinction between suicide attempt (SA) and non-suicidal self-harm (NSSH) is lacking. Although NSSH is a risk factor for SA, we do not currently know whether these behaviours lie on a continuum of severity, or whether they are discrete outcomes with different aetiologies. We conducted this exploratory genetic epidemiology study to investigate this issue further.MethodsWe explored the extent of genetic overlap between NSSH and SA in a large, richly-phenotyped cohort (the Avon Longitudinal Study of Parents and Children; N = 4959), utilising individual-level genetic and phenotypic data to conduct analyses of genome-wide complex traits and polygenic risk scores (PRS).ResultsThe single nucleotide polymorphism heritability of NSSH was estimated to be 13% (SE 0.07) and that of SA to be 0% (SE 0.07). Of the traits investigated, NSSH was most strongly correlated …
Smoking and the risk for bipolar disorder: evidence from a bidirectional Mendelian randomisation study
Authors
Jentien M Vermeulen,Robyn E Wootton,Jorien L Treur,Hannah M Sallis,Hannah J Jones,Stanley Zammit,Wim van den Brink,Guy M Goodwin,Lieuwe De Haan,Marcus R Munafò
Journal
The British Journal of Psychiatry
Published Date
2021/2
BackgroundThere is increasing evidence that smoking is a risk factor for severe mental illness, including bipolar disorder. Conversely, patients with bipolar disorder might smoke more (often) as a result of the psychiatric disorder.AimsWe conducted a bidirectional Mendelian randomisation (MR) study to investigate the direction and evidence for a causal nature of the relationship between smoking and bipolar disorder.MethodWe used publicly available summary statistics from genome-wide association studies on bipolar disorder, smoking initiation, smoking heaviness, smoking cessation and lifetime smoking (i.e. a compound measure of heaviness, duration and cessation). We applied analytical methods with different, orthogonal assumptions to triangulate results, including inverse-variance weighted (IVW), MR-Egger, MR-Egger SIMEX, weighted-median, weighted-mode and Steiger-filtered analyses.ResultsAcross …
Mapping drug epidemiology futures
Authors
Sarah Larney,Hannah Jones,Tim Rhodes,Matthew Hickman
Journal
International Journal of Drug Policy
Published Date
2021/8/1
Epidemiology is a core discipline generating evidence to inform and drive drug policy. In this essay, we speculate on what the future of drug epidemiology might become. We highlight for attention two areas shaping the future of drug epidemiology: nesting epidemiology within a ‘syndemic’ and ‘relational’ approach; and innovating in relation to causal inference in the face of complexity. We argue that shifts towards a more relational approach emphasise contingency, including in relation to how drugs might constitute benefit or harm. This leads us to speculate on a ‘positive epidemiology’; one that is configured not merely in relation to harm but also in relation to the potential benefits of drugs in relation to well-being. In responding to the complex challenges of delineating contingent causalities, we emphasise the potential of carefully conducted observational study designs that go beyond statistical associations to test …
Genetic studies of psychosis
Authors
Hannah J Jones,Stanley Zammit,James TR Walters
Published Date
2020/1/1
In this chapter we summarize a broad range of research studies that aim to improve our understanding of the genetic risk and etiology of psychosis. We begin by discussing early, mainly unsuccessful, attempts at identifying causal genetic factors through candidate gene and linkage studies. We then highlight the major advances achieved over the last decade by genome-wide association studies, including identification of common risk loci, quantifying genetic correlations between psychiatric disorders, and use of polygenic scores to explore phenotypic manifestation of genetic risk and for development of disease prediction models. We then summarize the main findings from copy number and rare variant studies and end with a discussion of the use of genetic factors in Mendelian randomization to improve causal inference.
Common mechanisms for type 2 diabetes and psychosis: Findings from a prospective birth cohort
Authors
Benjamin I Perry,Hannah J Jones,Tom G Richardson,Stan Zammit,Nicholas J Wareham,Glyn Lewis,Peter B Jones,Golam M Khandaker
Journal
Schizophrenia Research
Published Date
2020/9/1
BackgroundPsychosis and type 2 diabetes mellitus (T2DM) are commonly comorbid and may share pathophysiologic mechanisms. To investigate shared genetic variation and inflammation as potential common mechanisms, we tested: (i) associations between genetic predisposition for T2DM and psychotic experiences and psychotic disorder in young adults; (ii) the association between genetic predisposition for schizophrenia and insulin resistance (IR), a precursor of T2DM; and (iii) whether these associations are mediated by childhood inflammation.MethodsPsychotic experiences (PEs), psychotic disorder and IR were assessed at age 18. Polygenic risk scores (PRS) for T2DM and schizophrenia were derived based on large genome-wide association studies. Associations between PRS and psychotic/IR outcomes were assessed using regression analysis based on 3768 ALSPAC birth cohort participants with …
A population-based cohort study examining the incidence and impact of psychotic experiences from childhood to adulthood, and prediction of psychotic disorder
Authors
Sarah A Sullivan,Daphne Kounali,Mary Cannon,Anthony S David,Paul C Fletcher,Peter Holmans,Hannah Jones,Peter B Jones,David EJ Linden,Glyn Lewis,Michael J Owen,Michael O’Donovan,Alexandros Rammos,Andrew Thompson,Dieter Wolke,Jon Heron,Stanley Zammit
Journal
American Journal of Psychiatry
Published Date
2020/4/1
ObjectiveThe authors investigated the incidence, course, and outcome of psychotic experiences from childhood through early adulthood in the general population and examined prediction of psychotic disorder.MethodsThis was a population-based cohort study using the semistructured Psychosis-Like Symptoms Interview at ages 12, 18, and 24 (N=7,900 with any data). Incidence rates were estimated using flexible parametric modeling, and positive predictive values (PPVs), sensitivity, specificity, and area under the curve were estimated for prediction.ResultsThe incidence rate of psychotic experiences increased between ages 13 and 24, peaking during late adolescence. Of 3,866 participants interviewed at age 24, 313 (8.1%, 95% CI=7.2, 9.0) had a definite psychotic experience since age 12. A total of 109 individuals (2.8%) met criteria for a psychotic disorder up to age 24, of whom 70% had sought professional …
Comparison of Genetic Liability for Sleep Traits Among Individuals With Bipolar Disorder I or II and Control Participants
Authors
Katie JS Lewis,Alexander Richards,Robert Karlsson,Ganna Leonenko,Samuel E Jones,Hannah J Jones,Katherine Gordon-Smith,Liz Forty,Valentina Escott-Price,Michael J Owen,Michael N Weedon,Lisa Jones,Nick Craddock,Ian Jones,Mikael Landén,Michael C O’Donovan,Arianna Di Florio
Journal
JAMA psychiatry
Published Date
2020/3/1
ImportanceInsomnia, hypersomnia, and an evening chronotype are common in individuals with bipolar disorder (BD), but whether this reflects shared genetic liability is unclear. Stratifying by BD subtypes could elucidate this association and inform sleep and BD research.ObjectiveTo assess whether polygenic risk scores (PRSs) for sleep traits are associated with BD subtypes I and II.Design, Setting, and ParticipantsThis case-control study was conducted in the United Kingdom and Sweden with participants with BD and control participants. Multinomial regression was used to assess whether PRSs for insomnia, daytime sleepiness, sleep duration, and chronotype are associated with BD subtypes compared with control participants. Affected individuals were recruited from the Bipolar Disorder Research Network. Control participants were recruited from the 1958 British Birth Cohort and the UK Blood Service. Analyses …
Using genetic information to inform policy on cannabis
Authors
Lindsey A Hines,Jorien L Treur,Hannah J Jones,Hannah M Sallis,Marcus R Munafò
Journal
The Lancet Psychiatry
Published Date
2020/12/1
Cannabis is a commonly used drug, and a substantial minority of individuals who use it frequently will develop cannabis use disorder (CUD). Johnson and colleagues1 identify a high genetic correlation between liability to frequent cannabis use and CUD. This finding is in line with previous twin research, which has identified a high correlation between additive genetic influences acting on exposure to cannabis, frequency of cannabis use, and CUD. 2 One straightforward interpretation is that frequent use is on the causal pathway to CUD; however, Johnson and colleagues1 do not explore this formally (eg, by using Mendelian randomisation, which uses genetic variants as proxies for exposures of interest).In the present study and the previous twin study, a moderate proportion of genetic influences acting on liability to CUD appear to be unique from those acting on liability to frequency of use. These findings …
Omega-3 and Omega-6 fatty acids and risk of psychotic outcomes in the ALSPAC birth cohort
Authors
AD Thompson,HJ Jones,J Heron,J Hibbeln,S Sullivan,S Zammit
Journal
Schizophrenia research
Published Date
2020/10/1
BackgroundLong chain polyunsaturated fatty acid (PUFA) levels have been implicated in the pathology of psychotic disorders. We investigated the relationship between childhood PUFA levels and later psychotic experiences (PE's) in a large birth cohort.MethodsPlasma levels of Ω-3 and Ω-6 fatty acids (FA's) were assayed at ages 7 and 16 years. PE's were assessed at ages 12 and 18 years using a semi-structured interview. Primary outcome was any PE's at 18 years; sensitivity analyses examined incident PE's between ages 12 and 18 years, persistent PE's (at 12 and 18) and psychotic disorder at 18 years. Genetic instruments for Ω-3 and Ω-6 were derived and used in a multivariable Mendelian Randomization analysis.ResultsHigher levels of Ω-6 FA's AA, OA and AdA at age 7 years were weakly associated with a reduced risk for PE's at 18 years, however, effect sizes were small and attenuated after adjusting for …
Mendelian randomisation for nutritional psychiatry
Authors
Rebecca Carnegie,Jie Zheng,Hannah M Sallis,Hannah J Jones,Kaitlin H Wade,Jonathan Evans,Stan Zammit,Marcus R Munafò,Richard M Martin
Published Date
2020/2/1
Nutritional psychiatry is a growing area of research, with several nutritional factors implicated in the cause of psychiatric ill-health. However, nutritional research is highly complex, with multiple potential factors involved, highly confounded exposures and small effect sizes for individual nutrients. This Personal View considers whether Mendelian randomisation provides a solution to these difficulties, by investigating causality in a low-risk and low-cost way. We reviewed studies using Mendelian randomisation in nutritional psychiatry, along with the potential opportunities and challenges of using this approach for investigating the causal effects of nutritional exposures. Several studies have identified nutritional exposures that are potentially causal by using Mendelian randomisation in psychiatry, offering opportunities for further mechanistic research, intervention development, and replication. The use of Mendelian …
Evidence for causal effects of lifetime smoking on risk for depression and schizophrenia: a Mendelian randomisation study
Authors
Robyn E Wootton,Rebecca C Richmond,Bobby G Stuijfzand,Rebecca B Lawn,Hannah M Sallis,Gemma MJ Taylor,Gibran Hemani,Hannah J Jones,Stanley Zammit,George Davey Smith,Marcus R Munafò
Journal
Psychological medicine
Published Date
2020/10
BackgroundSmoking prevalence is higher amongst individuals with schizophrenia and depression compared with the general population. Mendelian randomisation (MR) can examine whether this association is causal using genetic variants identified in genome-wide association studies (GWAS).MethodsWe conducted two-sample MR to explore the bi-directional effects of smoking on schizophrenia and depression. For smoking behaviour, we used (1) smoking initiation GWAS from the GSCAN consortium and (2) we conducted our own GWAS of lifetime smoking behaviour (which captures smoking duration, heaviness and cessation) in a sample of 462690 individuals from the UK Biobank. We validated this instrument using positive control outcomes (e.g. lung cancer). For schizophrenia and depression we used GWAS from the PGC consortium.ResultsThere was strong evidence to suggest smoking is a risk factor for …
Hannah Jones FAQs
What is Hannah Jones's h-index at University of Bristol?
The h-index of Hannah Jones has been 21 since 2020 and 22 in total.
What are Hannah Jones's top articles?
The articles with the titles of
Precursors and correlates of transient and persistent longitudinal profiles of psychotic experiences from late childhood through early adulthood
Mendelian randomisation for psychiatry: how does it work, and what can it tell us?
Examining pathways between genetic liability for schizophrenia and patterns of tobacco and cannabis use in adolescence
Parental inflammatory bowel disease and autism in the offspring: Triangulating the evidence using four complementary study designs
Genetic liability to schizophrenia is associated with exposure to traumatic events in childhood
Causal associations between fatty acid measures and schizophrenia-a two-sample Mendelian randomization study
Childhood trauma as a mediator of the association between autistic traits and psychotic experiences: evidence from the ALSPAC birth cohort
Plasma polyunsaturated fatty acids and mental disorders in adolescence and early adulthood: cross-sectional and longitudinal associations in a general population cohort
...
are the top articles of Hannah Jones at University of Bristol.
What is Hannah Jones's total number of citations?
Hannah Jones has 2,619 citations in total.